付碧石，男，理学博士，教授，研究方向为宿主与病毒相互作用的分子机制。博士毕业后先后在佛罗里达州立大学和哈佛医学院开展博士后研究工作。长期从事病毒-宿主相互作用的蛋白质组学研究，获得了国际领先水平的重要研究成果。近五年的研究成果已第一作者或并列第一作者发表于Journal of Experimental Medicine，Nature Communication，PNAS，Cell Research，Plos Pathogen, Journal of Virology等国际学术期刊。其中JCR一区论文5篇，其他分区SCI论文4篇。参与和主持多项自然国家基金。

主要代表著作：

1.Bishi Fu * … Martin E. Dorf (2017). ZMPSTE24 defends against influenza and other pathogenic virus. Journal of Experimental Medicine.

2.Lingyan Wang*, Bishi Fu*… Shitao Li(2017). Dynamic and Comparative Influenza-Host Protein Interactomes Define the Role of PKP2 in Perturbation of Viral Polymerase Complex. Nature Communication.

3.Bishi Fu*…Martin Dorf (2015). TRIM32 senses and restricts influenza A virus by ubiquitination of PB1 polymerase. PLOS Pathogens.

4.Bishi Fu*… Fanxiu Zhu (2015). Activation of p90 Ribosomal S6 Kinases (RSKs) by ORF45 of Kaposi Sarcoma-Associated Herpesvirus is Critical for Optimal Production of Infectious Viruses. Journal of Virology.

5.Bishi Fu*…Martin E Dorf (2014). The ubiquitin conjugating enzyme UBE2L3 regulates TNFα-induced linear ubiquitination. Cell Research.